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Fueling Infant Gut Microbes
Monday, April 18, 2016
UC Davis researchers have shown that an enzyme produced by beneficial microbes in babies’ intestines is able to harvest specific sugar compounds from human breast-milk and cow’s milk.

New Clue In Lupus Autoantibody Production
Friday, April 15, 2016
A signaling molecule called interferon gamma could hold the key to understanding how harmful autoantibodies form in lupus patients.

New Method to Preserve Device to Monitor HIV Treatment
Thursday, April 14, 2016
Inspired by pregnancy tests, scientists have developed a method to store microfluidic devices for months without refrigeration, giving developing countries an inexpensive and reliable way to treat patients.

UCSF Immunologist to Head New Parker Institute for Cancer Immunotherapy
Thursday, April 14, 2016
Renowned UC San Francisco immunologist Jeffrey Bluestone, PhD, has been named president and CEO of the Parker Institute for Cancer Immunotherapy, a national initiative launched with a $250 million grant from The Parker Foundation.

Recreating a T-Cell Receptor Signaling Pathway
Wednesday, April 13, 2016
Successfully re-creating a T-cell receptor signaling pathway independent of the cell itself provides novel insights into how protein signaling works in a complex cellular process.

Investigating Kidney Cancer Therapies
Wednesday, April 13, 2016
Two drugs known to improve survival for patients with metastatic renal cell carcinoma do not reduce the risk of cancer recurrence when administered after surgery.

UCSD Scientists Receive NIH Grant to Combat Antibiotic Resistance
Wednesday, April 13, 2016
Interdisciplinary program will use systems biology approaches to understand how antibiotics work in concert with patient’s immune system.

NanoString, HalioDx Collaborate
Tuesday, April 12, 2016
Company has announced collaboration with HalioDx to jointly develop and commercialize novel gene expression assays in immuno-oncology.

CNS Inflammation: A Pathway and Possible Drug Target
Tuesday, April 12, 2016
Scientists have long known that the central nervous system (CNS) has a remarkable ability to limit excessive inflammation in the presence of antigens or injury, but how it works has been unclear.

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