Cellular Senescence: A Common Thread Across Age-Related Diseases
Listicle
Last Updated: July 7, 2021
Written by
Michele Trott, PhD
Michele Trott holds a PhD in endocrinology from Lincoln University. She works as a freelance science writer and scientific content manager for Izon Science.
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Though degenerative and hyperplastic diseases are diverse and manifest in very different ways, they appear to be connected by at least one common process – cellular senescence. Cellular senescence describes the complex cellular response to stress that includes irreversible arrest of the cell cycle, resulting in the prevention of defective or damaged cells.
Download this listicle to learn about four cellular senescence studies:
- The elimination of senescent cells delays tumorigenesis and extends lifespans in mice
- Senolytic treatment reduces plaque development and increases vasomotor function
- Implications in Alzheimer’s disease
- Senolytics for kidney disease: a promising preliminary clinical report
Meet the Author
Michele Trott (nee Wilson) is a New Zealand-based science writer who pursues freelance writing projects alongside her in-house role as a scientific content writer at Izon Science. After completing her PhD (Endocrinology) at Lincoln University (NZ), Michele began pursuing her science writing career with Technology Networks in the UK. There, she spent 12 months covering a maternity leave role as a science editor, and managed the cell science, biopharma and diagnostics communities. Upon her arrival back in NZ, Michele established “Choice Science Writing” and became a full-time freelance science writer, working with life science marketing agencies, biotech companies – and of course, Technology Networks – to produce engaging articles and blog posts. In April 2021, she took up the opportunity to join the team at Izon Science, a company that enables the precise separation and analysis of extracellular vesicles and other nanoparticles.
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