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Dementia Protein Stops Tau Being Cleared From the Brain

News   Feb 19, 2020 | Original story from the University of South Florida

 
Dementia Protein Stops Tau Being Cleared From the Brain

A beta-arrestin-2 oligomer (foreground image including white) shown in a nerve cell (background). Oligomerized beta-arrestin-2 plays a central role in impairing tau clearance and the development of tau aggregates (magenta) in frontotemporal lobe degeneration and Alzheimer's disease. Credit: Cynthia Greco (artist) and Eric Lewandowski (beta-arrestin-2 protein modeling), University of South Florida Health

 
 
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