This Week on NeuroScientistNews: 4 May – 8 May
News May 08, 2015
Neuroscience beyond the bench; link between diabetes and Alzheimer’s; organization of the sensory cortex, and more.
Not all neuroscientists work at the lab bench. In this profile we follow the story of Won Yung Choi, National Sales Manager for the Americas, Bitplane. Choi discusses her experiences transitioning out of academia to a non-traditional neuroscience role.
Although the use of cannabis as a medical drug is currently booming, we should not forget that leisure time consumption -- for example, smoking weed -- can cause acute and chronic harms. These include panic attacks, impaired coordination of movement, and nausea, as Eva Hoch and colleagues show in a topical review article in Deutsches Ärzteblatt International.
Researchers have uncovered a unique connection between diabetes and Alzheimer's disease, providing further evidence that a disease that robs people of their memories may be affected by elevated blood sugar, according to scientists at Washington University School of Medicine in St. Louis.
Researchers at the Max Planck Institute for Biological Cybernetics (Germany), VU University Amsterdam (Netherlands) and Max Planck Florida Institute for Neuroscience (USA) succeed in reconstructing the neuronal networks that interconnect the elementary units of sensory cortex – cortical columns.
Researchers at the University of North Carolina (UNC) at Chapel Hill have used exosomes -- tiny bubbles of protein and fat produced naturally by cells -- to bypass the body's defenses and deliver a potent antioxidant directly to the brain to treat Parkinson's disease.
3-D Representation of Motor Space in the BrainNews
Neuroscientists found that the superior colliculus, a region of the midbrain, is where three-dimensional motor space is mapped, and importantly they showed how the activity of neurons in this brain region leads to controlled spatially targeted movement.READ MORE
Mouse Mutation Protects Against Alzheimer'sNews
Researchers have discovered a mutation that can protect against Alzheimer’s disease in mice. They found that a specific mutation can reduce the characteristic accumulation of the amyloid-beta peptide that occurs in the nerve cells of Alzheimer's patients.
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